Hosted by Dr. Michael Schlossmacher
Learning Objectives
1. Abrogating physiological a-synuclein tetramers produces neuropathology and a progressive motor syndrome resembling PD, including partial protection by female sex based on synaptic estrogen receptor availability
2. Distinct a-synuclein mutations causing graded solubility can impact synaptic function and motor skill learning abilities
3. Increasing certain membrane phospholipid saturation improve the a-synuclein physiological state in vivo and may guide the development of PD therapeutic approaches.